房屋建筑材用防腐剂的毒性试验

通过对十八种药剂配方(其中水溶性配方15种、油溶性配方3种)对木腐菌的毒性试验及对初筛后的配方进行流失后毒性试验,结果表明,水溶性配方中的铜铬砷即CCA_3、CCA_4、CCA_5,铜铬硼即CCB_5配方,以及油溶性配方中的P—B剂的毒性较大。     

白僵菌优良菌株的选育

本研究将所收集的28个菌株,用稀释单孢分离的方法,进行生长特征、生长速度、产孢量和室内外对松毛虫致病力的测定比较,以闽林2、闽林1、闽林3号菌株表现突出,为优良菌株。     

铝胁迫对山茶属植物光合特性的影响

以山茶属植物山茶花（Camellia japonica）品种花克瑞墨大牡丹以及茶（Camellia sinensis）的2个品种（白茶、乐茶）为材料，设置不同铝处理浓度，研究了铝对山茶属植物叶片光合作用特性的影响。结果表明，低浓度的铝处理促进了山茶属植物叶片叶绿素的合成和净光合速率的升高，高浓度的铝处理抑制了山茶属植物叶片叶绿素的合成，降低了净光合速率，胞间CO2浓度在不同铝浓度处理下变化不大，说明低浓度的铝能促进山茶属植物的光合作用，高浓度铝抑制了山茶属植物的光合作用。     

杉木根际与非根际土壤酶活性比较

杉木根际与非根际土壤酶活性比较陈ｆ竣（中国林业科学研究院林业研究所北京１０００９１）李传涵（华中农业大学土化系武汉４３００７０）关键词杉木,根际土壤,土壤酶,杉木中毒由于林木生长的影响,使得林木根际土壤ｐＨ值、养分含量、微生物数目、生化活性等性质发生...     

Growth and nutritional responses of bareroot Jeffrey pine on a Sierra Nevada surface mine to minisite applications of fertilizer and lime

The effects of two controlled release fertilizers, Forestcote 22-4-6 + Minors and Dry Site 21-6-2 + Minors, and also dolomitic lime, on growth and nutrition of bareroot Jeffrey pine (Pinus jeffreyi Grev. & Balf.) were investigated. Forestcote, which consists of loose prills, and Dry Site, consisting of prepackaged prills, were administered with two rates, while a single liming rate was used. An acidic Sierra Nevada surface mine provided the study site, and all amendments were applied at outplanting to the backfill of augered planting holes. Without impacting survival, both fertilizer formulations enhanced seedling growth, particularly at a 16 g rather than an 8 g application rate, although Forestcote was marginally more effective in this regard. In contrast, liming at a 27 g rate induced severe mortality and offset much of the growth gains resulting from fertilization. Improvements in N and P nutrition, as revealed by foliar analysis, probably accounted for much of the growth stimulation exhibited by fertilized seedlings, but reductions in the uptake of potentially phytotoxic trace elements such as Mn and Al and increases in base cation/metallic element ratios likely also contributed. The high application rate was generally preeminent in eliciting these nutritional responses. Liming induced little alteration of seedling nutrition. Impaired water relations may have caused the unfavorable responses to this treatment.     

铝盐对厌氧微生物产甲烷活性的影响研究

废水中铝盐含量增高以及铝盐的生物毒性使其对废水生物处理工艺中微生物产生的危害引起了人们的关注。本文以产甲烷活性为指标，通过投加氯化铝测定其对颗粒污泥的产甲烷毒性来研究铝盐对厌氧菌的危害。结果表明，铝盐使厌氧颗粒污泥的活性明显降低，但其毒性可以驯化，且清除铝盐后厌氧颗粒污泥的活性可以恢复。     

长链脂肪酸对厌氧颗粒污泥产甲烷活性的影响及其相互作用研究

采用间歇培养实验研究了辛酸、癸酸和月桂酸等三种长链脂肪酸对UASB反应器厌氧颗粒污泥产甲烷活性的毒性，以及癸酸对辛酸和月桂酸抑制作用的影响。结果表明，辛酸、癸酸和月桂酸对UASB反应器厌氧颗粒污泥的产甲烷活性有严重抑制，对厌氧颗粒污泥最大比产甲烷活性产生50%抑制的浓度(IC     

基于WASP模型的东辽河水质模拟研究

借助数学方法和现代计算机技术,根据东辽河的水流特性、水文、污染源的调查资料及水质实测资料,选用USEPA研发并推荐使用的水质分析模拟程序WASP的最新版本WASP7．3,定量描述排入河流中污染物的数量与河水水质之间的关系,分析和掌握东辽河2009年主要污染物（氨氮）的时空变化规律,结果表明：WASP7．3模型在东辽河流域具有较强的适用性。从年内变化特征看,在枯水期各段氨氮浓度最大,其次是丰水期,平水期最小;从空间分布特征看,在第8、9和第10段（辽源市下游）氨氮超标严重。对模拟结果的分析表明点源和非点源污染物向河流的排放和降水量的减少是东辽河水质恶化的重要原因。研究结果为改善东辽河水环境质量及保护流域生态系统提供科学依据。     

Sphingosine-1-phosphate receptor-2 attenuates lipopolysaccharide-induced acute lung injury

AIM: To investigate the effects and mechanisms of sphingosine-1-phosphate receptor-2 (S1P2R)on lipopolysaccharide (LPS)-induced acute lung injury (ALI). METHODS: ALI model was induced by intratracheal administration of LPS in both wild-type mice and S1P2R -deficient mice. The pathological changes in the lung tissues were observed, and the protein concentration, total cell number, neutrophil ratio, TNF-α level and IL-6 level were determined in the bronchoalveolar lavage fluid (BALF) 24 h after LPS injection. In order to investigate the mechanisms of S1P2R in LPS-induced ALI, 10 min before LPS injection, both wild-type mice and S1P2R -deficient mice were injected with nitric oxide synthase inhibitor by tail vein injection, the pathological changes of the lung tissues were observed, and the protein concentration and total cell number in BALF were determined 12 h after LPS injection. RESULTS: Compared with wild-type mice, S1P2R -deficient mice showed more severe LPS-induced ALI, and the protein concentration, neutrophils and inflammatory cytokines in BALF were significantly increased in S1P2R -deficient mice. Administration of nitric oxide synthase inhibitor N^ω-L-nitro-arginine methyl ester protected S1P2R -deficient mice from aggravation of ALI. CONCLUSION: S1P2R mediates the protection from LPS-induced ALI possibly through inhibiting nitric oxide synthase.     

Mesenchymal stem cell-induced CD8α+ Jagged2high CD11bhigh regulatory dendritic cells prevent and treat mouse aGVHD

AIM: To investigate the role of mesenchymal stem cell-induced regulatory dendritic cells (MSC-DCregs) in mouse acute graft-versus-host disease(aGVHD) model. METHODS: Bone marrow cells from BALB/c (H-2^d) mice were isolated and were induced to differentiate into DCs. The DCs were selected by flow cytometry, and after 10 d co-culture with MSCs, they were induced to be MSC-DCregs. Male 8-week-old C57BL/6 (H-2^b) mice were used as donor mice. The female 8-week-old BALB/c (H-2^d) mice, who had received 100 cm source-skin distance, 30 cm×30 cm radiation field, 700 cGy total body irradiation (TBI) pretreatment were used as recipient mice. The recipients were divided into 5 groups: control group, TBI group (injected with medium only), bone marrow transplantation group (injected with 1×10⁷ bone marrow cells), aGVHD group (injected with 1×10⁷ bone marrow cells and 1×10⁷ spleen cells), and MSC-DCregs group (injected with 1×10⁷ bone marrow cells, 1×10⁷ spleen cells and 1×10⁶ MSC-DCregs). The white blood cell count, recipients' chimerism, clinical evaluation of aGVHD, survival analysis and pathological changes were determined. RESULTS: Hematopoieic recovery was seen at 10 d after transplantation. The recipients' chimerism was parallel to the donors' at 30 d. The median survival time of the mice in aGVHD group and MSC-DCregs group was 27 d and 33 d, and the survival rates at 30 d were 20% and 100% (P<0.01), respectively. The clinical scores of the mice in MSC-DCregs group were lower than those in aGVHD group (P<0.01). Moreover, the pathological changes in the skin and liver of the mice in MSC-DCregs group were less serious than those in aGVHD group. CONCLUSION: The MSC-DCregs induce an aGVHD tolerance in vivo, and further research of its mechanism is still in great necessary.